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If this proves up it is going to be an absolute disaster for everyone in the health field -- in fact, it will be as bad as the disaster that resulted from exposure of the fact that leeches were worthless in "treating disease."
Coronary Artery Disease, or "CAD" for short, has proceeded from the following general premises:
1. Cholesterol in the blood binds to sites of inflammation in the artery, treating it much like it would a splinter in your foot.
2. That process is cumulative and, over time, blocks more and more of the artery.
3. Eventually this produces a blockage that either breaks off (a stroke) or you have a heart attack due to insufficient blood delivery to the heart muscle.
In fact if you look around Google for "Coronary Artery Blockage image" you will find hundreds of images that look like this:
There's one problem -- they're all wrong.
The paper cited above points out the logical fallacies that have attached to the "common model" you're taught and that your doctor -- and cardiologist -- is using.
Now let's add some more facts to the mix that are discussed in the paper:
This paper roundly demolishes a number of claims that are made every day in the media and the medical establishment, and what's worse is that it completely decimates the balance of benefit and harm argument for the use of drugs to "lower cholesterol" such as statins because once the inner coronary arterial wall has become vascularized LDL-C will inevitably be picked up and accumulated.
Let's put forth what is directly attacked and, if this paper holds up, refuted:
The problem with what's presented here, from a medical perspective, is that if this paper proves up it doesn't leave you with a trillion dollar industry you can force people to spend money on by scaring them to death.
If in fact the growth of the inside layer of your coronary arteries in thickness is a risk factor that simply comes with time on the planet then given the inherent response of cells that become less-than-well supplied by oxygen (that is, to stimulate vascularization) there may be no actual cure available for this condition; it may simply be inherent in getting older, with some people more susceptible than others.
If, on the other hand, there is some sort of systemic insult responsible for the thickening of these inside walls beyond the limits of diffusion to provide transport that might be able to be addressed. The problem with such a belief is that you then have to find the same insult pathway -- whether from the same cause or not -- in the other mammals that also suffer from the condition. That becomes a rather serious problem since obviously other large placental mammals don't eat trans-fats or refined sugars, as just one example.
In short while this paper is a not a smoking gun as to cause it refutes a lot of false claims about coronary arterial disease and, if it holds up to scrutiny you may be witnessing the imminent destruction of a trillion-dollar a year scam.
PS: This paper's description is also consistent with why smoking promotes (but does not cause) coronary artery disease. Smoking raises CoHb levels, which in turn would tend to lead to cellular hypoxia, with the most-severe effects being in cells where the diffusion path is the longest. That, in turn, would be expected to promote vascularization..... Likewise, it explains the association between sleep apnea where depression of oxygen saturation has been documented and CAD -- an association that has been shown but I've yet to see a plausible scientific explanation as to why they're associated. This definitely appears to merit further study!
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